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Therefore, while the GM represents a key modulator of bone homeostasis, its precise function and its contribution are likely conditioned by the predominant underlying etiology of bone loss and the specific skeletal site examined

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Mnzel T, Gori T, Keaney JF Jr, Maack C, Daiber A (2015) Pathophysiological role of oxidative stress in systolic and diastolic heart failure and its therapeutic implications