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A putative transporter, encoded by gliA , has been shown to facilitate gliotoxin efflux, and increased tolerance to exogenous gliotoxin, when expressed in Leptosphaeria maculans [13]

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HIV protease inhibitorsritonavir, indinavir, saquinavir, nelfinavir, and zidovudinewere described as ABCC1 substrate [35], suggesting that the overexpression of ABCC1 in infected cells makes these cells more resistant to chemotherapy

The GLP-1R agonist liraglutide limits hepatic lipotoxicity and inflammatory response in mice fed a methionine-choline deficient diet

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