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Cunningham GR, et al

Furthermore, cells with the knockout of the ASCT2 (SLC1A5) transporter demonstrated a high adaptation to amino acid imbalance via the upregulation of redundant transporters such as ASCT1 (SLC1A4) and SLC38A2 (SNAT2) [14,17]

On top of its important role as the powerhouse of the cell, these organelles are also essential regulators of cellular metabolism, redox state, intracellular calcium signaling, and programmed cell death mechanisms [12,13,14,15]

Also Read: How To Manage Vitamin D Deficiency in Children How to Lower B12 Levels If youre wondering, What is the treatment for high vitamin B12? it typically involves identifying and managing the underlying cause rather than reducing the vitamin levels directly