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The discovery of SQORs unique cysteine trisulfide bond cofactor explains its remarkable catalytic efficiency ( 2 S levels, keeping them within an optimal physiological range: on the one hand, genetic defects (e.g., in Leigh syndrome) or CoQ deficiency that abolish SQOR activity can cause toxic H 2 S accumulation, inhibiting the mitochondrial respiratory chain and leading to severe energy failure

Biological treatment strategies for disc degeneration: potentials and shortcomings

Eales, J

Near the top are large randomized controlled trials, the gold standard for determining whether a therapy truly works and is safe across diverse populations

In neurological disorders, both oxidative stress and mitochondrial damage can contribute to ferroptosis, resulting in nerve cell dysfunction and death