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By upregulating and activating SIRT1, which subsequently deacetylates and functionally inhibits STAT3, GHK-Cu entirely short-circuits the inflammatory cascade, promotes profound mucosal healing, and restores the integrity of tight junction proteins (such as ZO-1 and Occludin) within the cellular barrier

It plays a critical role in the synthesis of myelin, protecting nerve fibers and ensuring efficient nerve signal transmission

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