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Defects in components of the sex determination pathway result in mutants that fail to differentiate into mature gametes appropriately 18,19

Abnormalities in the mitochondrial electron transport chain (ETC), combined with impaired antioxidant defenses, lead to excessive accumulation of reactive oxygen species (ROS), which damage lipids, proteins, and mitochondrial DNA, ultimately promoting tubular apoptosis, interstitial fibrosis, and glomerulosclerosis (26, 27)