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These results suggest that metformin promotes GLUT1 expression through TXNIP suppression, independently of AMPK, and probably via mitochondrial complex I inhibition

Its being studied for GERD because preclinical research shows it accelerates mucosal healing in the stomach and esophagus by upregulating VEGF and FGF, promotes angiogenesis at injury sites, and modulates inflammatory pathways implicated in reflux esophagitis

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So all of these things, which we don't really know about how to manage in the gut is probably going to explode my guess in the next several decades because that, that's where I think a lot of people's health is sort of

Time NAC with a meal