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Astragaloside IV inhibits oxidative stress-induced mitochondrial permeability transition pore opening by inactivating GSK-3 via nitric oxide in H9c2 cardiac cells

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As the score value of the Si-DSS group was significantly lower than that of the Con-DSS group, it was confirmed that the injury of both the colon and rectum was suppressed in the Si-DSS group (Fig

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Activation of TRPV1 attenuates high salt-induced cardiac hypertrophy through improvement of mitochondrial function