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These medications work by slowing down digestion (delaying gastric emptying)

GLP-1R activation exerted antifibrotic effects by blocking the interaction between the NLPR3 inflammasome and PFKFB3-driven glycolysis, leading to the inhibition of lactate production and subsequent histone lactylation, and thereby decreasing profibrotic gene expression in fibroblasts under inflammatory stress

As illustrated in Figure 1, this integrated strategy demonstrates multi-target synergistic effects, significantly improving clinical response, endoscopic remission, and mucosal healing rates, while exhibiting a unique efficacy enhancement and toxicity reduction profile

To preserve the quality of oocytes, it is essential to maintain proper lipid oxidation with or without minimal production of free radicals [46]