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In multiple preclinical models, pharmacological inhibition of ferroptosis preserves myocardial and vascular function in settings such as ischemic injury, heart failure, and atherosclerosis, positioning ferroptosis as both a central disease driver and a potential therapeutic target

In: De Saeger S., editor

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In this way, glutathione acts as a protective shield, ensuring that harmful substances do not accumulate and cause further damage to cellular structures

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