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The clinical features strongly resemble classical EDS (cEDS) with two key differences: (1) the absence of atrophic scarring and (2) autosomal recessive inheritance

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BPC-157s primary signalling mechanisms are well-characterised: it activates VEGFR2, drives downstream ERK1/2 phosphorylation, and stimulates nitric oxide synthesis via the Akt-eNOS axis producing pro-angiogenic, fibroblast-activating, and extracellular matrix-stabilising effects in experimental models

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For elbow tendinopathy, local injection near the affected area delivers the highest concentrations directly to damaged tissue