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This creates a vicious, self-perpetuating cycle: mitochondrial damage causes oxidative stress, the lack of methylation prevents the body from neutralizing that stress, and the resulting inflammation further damages the mitochondria and endocrine system

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Histopathological findings revealed preservation of neural architecture, with reduced axonal degeneration, diminished neuronal loss, and limited formation of cystic cavities, suggesting an attenuation of secondary injury processes (12)