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This may be attributed to the role of metformin in reducing the production of sFlt-1 and soluble endoglin by endothelial cells, trophoblasts, and syncytiotrophoblasts, and modulating anti-angiogenic factors at the mitochondrial level (Romero et al., 2017)
Following a similar approach, it was demonstrated that a decreased level of SFN results in neuroinflammation and oxidative stress, as indicated by lower concentrations of malondialdehyde, TNF- , and IL-1, respectively (85)
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