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Aging impairs these stress-responsive axes, which weakens EC adaptation to mechanical stress and promotes dysfunction

During proline catabolism, electrons derived from PRODH activity are transferred to the mitochondrial ETC, potentially supporting the regeneration of ubiquinol (CoQ 10 H 2 )

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The inclusion of this compound in research protocols introduces a potent variable for investigating anti-inflammatory signaling and the prevention of muscle catabolism at the cellular level