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Abnormalities in the mitochondrial electron transport chain (ETC), combined with impaired antioxidant defenses, lead to excessive accumulation of reactive oxygen species (ROS), which damage lipids, proteins, and mitochondrial DNA, ultimately promoting tubular apoptosis, interstitial fibrosis, and glomerulosclerosis (26, 27)

Scott Butsch, MD

Sure-I will have a bite

Despite decades of research, DSIP remains something of a scientific enigma: no specific receptor has been conclusively identified, no precursor protein gene has been isolated, and its precise mechanism of action continues to be investigated

The deal comes after Regenerons pipeline suffered a body blow last week on mixed results for its Sanofi-partnered Dupixent follow-up