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Bioavailability is usually determined by calculating the respective plasma drug exposure assessed as the total area under the drug plasma concentration versus time curve (AUC) after oral and intravenous administration as: In general, determinants of oral drug bioavailability include fraction of dose absorbed in the gastrointestinal tract (GIT) and fraction of dose that escapes elimination by the intestinal tract, liver, and lung

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Arginine salt (arginate) form shows dramatically improved oral bioavailability (over 90% versus about 3% for acetate) due to enhanced gastric stability

The inclusion of benzyl alcohol helps inhibit microbial growth after initial vial entry, supporting controlled multi-use handling within laboratory environments

For these reasons, several drugs have been explored for their protective effects in oxidative stress in vitro models and ischemic stroke mouse models (middle cerebral artery occlusion/reperfusion)