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Boiga melanota), separate from Boiga dendrophila
van den Beld AW, de Jong FH, Grobbee DE, Pols HA, Lamberts SW
On the one hand, in some in vitro or animal models, enhanced BCAA catabolism has been reported to upregulate certain M2-related markers and support alternative activation programs, whereas inhibition of BCAA metabolism may impair the establishment of M2-like phenotypes ( At the molecular level, the coupling of BCAAs with mTORC1 signaling provides a key clue for understanding their pro-inflammatory effects
doi: 10.1016/j.fertnstert.2009.10.046 150 KamkarNRamezanaliFSabbaghianM
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