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Psychiatry 22 , 12821290 (2017)

Finally, excessive glucose flux through the hexosamine pathway leads to UDP-GlcNAc accumulation and subsequent O-GlcNAcylation of serine/threonine residues on antioxidant enzymes, like SOD, reducing their activity (Paneque et al., 2023)

10.3390/molecules22081303 159 NiuC.PangG

In flies, loss of Shawn, the SLC25A39 homolog causes neurodegeneration and synaptic defects 26