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Research has demonstrated that NCOA4 knockdown reduces intracellular iron levels and lipid peroxidation, thereby attenuating zinc nanoparticle-induced cell death (70)

Mitochondrial Protection : Concentrates within mitochondria to support these cellular health powerhouses, helping maintain healthy oxidative balance and supporting overall energy metabolism

This phenotype was confirmed by targeted measurement of triglycerides in serum (Fig

Other pathways implicated, explaining the development of vitiligo, include autophagy and ferroptosis, where oxidative stress impacts the cells ability to clear damaged components, eventually leading to cell death and depigmentation 17,46

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