Concluding, whatever the myotendinous junction lesions current therapy (i.e., biological scaffolds, administration of active compounds, electrospinning, self-reorganized constructs (for review see, i.e., [98])), there is still the unresolved problem of the myotendinous junction and bone-tendon junction as extremely specialized tissues, inadequate repair through mechanically and histologically suboptimal scar tissue, decreased functional properties, and greater risk of recurrent injury [98]
By providing exogenous pyridoxine that is readily converted to PLP, the injection corrects the coenzyme deficiency and reactivates these metabolic pathways
There are capsules, creams, and nasal sprays
ACS Appl
Claudio Nicoletti, University of Florence, Italy Updates Copyright 2021 Vanuytsel, Tack and Farre