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C-terminal amidation, as seen in Nle-Tyr-Ile-His-NH, also provided a more modest increase in stability

-cell-selective knockout of the GLP-1R in mice with impaired glucose tolerance after intraperitoneal injection of glucose and exogenous supplementation of GLP-1 did not induce insulin secretion, but glucose tolerance returned to normal after oral glucose, suggesting that the extra-islet GLP-1R plays a regulatory role in oral glucose tolerance (Smith et al., 2014)

Hyperinsulinism-hyperammonemia syndrome: a de novo mutation of the GLUD1 gene in twins and a review of the literature

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While the liver has substantial storage capacity, supraphysiological doses (such as from injections) are largely excreted in the urine