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For communicating the extent of HPHCs emitted by our smoke-free products, we use reduced emission statements, which indicate the relative reductions of a selected number of toxicants in the emissions of smoke-free products compared with cigarettes
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Persistent mitochondrial damage in airway epithelium of COPD patients is further evidenced by the increased expression of PINK1, a sensor for damaged mitochondria, and PPARGC1, a regulator of mitochondrial biogenesis [20, 46]
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